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Research has examined its effects on c-Met receptor activation, downstream kinase signaling networks, dendritic architecture regulation, and molecular processes associated with synaptic structure and connectivity

Unlike true peptides, Dihexa is not degraded by peptidases, has a relatively small molecular weight (~480 Da), and crosses the blood-brain barrier efficiently after systemic administration

9.0% weight loss, with an estimated treatment difference of 1.8% [1]

from an independent Chinese research group (PMID: 34827486) demonstrated that Dihexa rescued cognitive impairment in APP/PS1 Alzheimer's disease model mice via the PI3K/AKT signaling pathway, providing the first independent replication of cognitive enhancement effects

Additional study is necessary to provide a comprehensive understanding of its safety profile and long-term consequences