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Through this network-oriented reevaluation, we aim to provide a unifying theoretical perspective that organizes fragmented observations, highlights key regulatory hubs, and generates testable predictions to guide future basic research and the development of potential clinical intervention strategies

This matters because thyroid hormones control testosterone production in several ways: They trigger gonadotropin-releasing hormone (GnRH) release They make Leydig cells more sensitive to luteinizing hormone (LH) They boost the activity of testosterone-making enzymes Studies show that low thyroid function often leads to less testosterone

Biphasic action of iodine in excess at different doses on ovary in adult rats

"The role of the one-carbon cycle in the developmental origins of Type 2 diabetes and obesity"

Similarly, supplementation with arachidonic acid or other PUFAs can increase cellular sensitivity to ferroptosis ( Figure 2 Lipoxygenases (ALOX) are non-heme iron-containing oxygenases that can directly oxidize PUFAs in cellular membranes to produce lipid peroxides, suggesting that ALOXs may mediate the occurrence of ferroptosis (36)