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In hepatocellular carcinoma models, this approach produced high levels of ONOO inside mitochondria, causing irreversible damage to the ETC, inhibiting ATP production, and triggering cancer cell death, which significantly reduced tumor volume (134)

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This technique takes advantage of the high affinity of many proteins for specific chemical groups

Together, these clinical human data can join the large range of beneficial effects of the BPC 157 therapy indicated by the animal experiments

iPSC-MSCs acquire a rejuvenated status and exhibit a longer lifespan compared to primary MSCs [273,274,275]