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In the heart under pressure overload, intracellular copper accumulation can trigger cardiomyocyte cuproptosis via FDX1 activation, leading to lipoylated protein aggregation and mitochondrial damage, thereby exacerbating cardiac injury [12]

Progressive degeneration of nigrostriatal dopamine neurons following intrastriatal terminal lesions with 6-hydroxydopamine: a combined retrograde tracing and immunocytochemical study in the rat

In radial glia, SVCT2 becomes apically polarized by incorporating AA from the CSF

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