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Additionally, the quinone structure of DOX promotes oxidative stress within cancer cells by participating in redox cycling 1,2

113, E3745E3754

10.1073/pnas.1400102111 Proc

These hypotheses have sprung from its perceived potential to stimulate the production of collagen, elastin, and glycosaminoglycans, crucial intercellular matrix components

LIP and Fe2 + formed by the degradation of ferritin can participate in the intracellular Fenton reaction, which is involved in oxidative stress (Figure 2)