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A series of recent studies implicated sirtuins as potent negative regulators of fibroblast activation and myofibroblast differentiation, and sirtuin dysfunction has been linked to the pathogenesis of SSc and other fibro-inflammatory conditions 28,29,38,39,40,41,42

These include catalase (CAT), superoxide dismutase (SOD) and glutathione peroxidase (GPx), as well as a methemoglobin reductase activity that reduces methemoglobin back to normal ferrohemoglobin

Led by Anders Fink-Jensen at the University of Copenhagen, this work explored the use of exenatide

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In Lawrence et al., ASA induced a significant reversible fall in serum folate