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Deficiency of SLC25A39 in LUAD cells leads to disrupted mitochondrial function and activated apoptosis To further elucidate the functional mechanisms of SLC25A39 in LUAD, we analyzed SLC25A39 expression in The Cancer Genome Atlas (TCGA) database and identified the top 500 genes most strongly correlated with SLC25A39

PubMed,

Hercus et al, 2003

In contrast, the eye lens lacks vasculature and is therefore not exposed to inflammatory changes in the same way

Additionally, the study delved into the possible role of the FAK-paxillin pathway (a pair of focal adhesion-linked proteins that relay signals following integrins) in conveying the action of BPC-157