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Another study reported that a humanmouse monoclonal antibody binding to methylphenidate, named anti-METH antibody (mAB7F9), elicited a significant reduction in the METH concentration ( Drugs for Chronic Damage Long-term exposure to METH leads to DA depletion, impairment of dopaminergic markers, and neuronal degeneration ( cis -retinoic acid (9cRA), a biologically active derivative of Vit.A, reversed the METH-induced decrease in immunoreactivity and apoptosis of T-helper cells via inhibition of the export of nuclear receptor 77 from the nucleus to the cytosol in primary cultured dopaminergic neurons

Specifically, this work is the first to propose and examine the modulation of endothelial-mesenchymal transition (EndMT) by hydrogen as a mechanistic basis for improved drug delivery and disease outcome in silicosis

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