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Neuropathological investigations in the substantia nigra pars compacta of Parkinsons disease patients demonstrate that deficient PGC-1 expression compromises mitochondrial biogenesis and disrupts the coordinated transcriptional activation of antioxidant stress-responsive gene networks, thereby precipitating dopaminergic neuronal degeneration (Piccinin et al., 2021)

Conclusion This study confirms the previous results that epitalon increases telomere lengths in normal epithelial and fibroblast cells through the up-regulation of telomerase

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These findings could be explained by the difference in dilution between the two samples as Petrochenko et al