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The primary downstream pathway involves JAK2 trans-phosphorylation and subsequent activation of STAT5a/b, which drives transcription of key targets including hepatic IGF-1, IGFALS, and feedback regulators CISH and SOCS2

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This is consistent with many animal studies and in-depth immunological studies that show, as expected, a measurable contribution to protection via non-nAb [56,57,58], NK cells [59], and MAIT cells [60, 61] and in particular T cells [62,63,64,65]

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