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J Clin Investig 127:28552867

Finally, we address key translational challenges, including dosing constraints, compensatory mechanisms, and SLC6 family selectivity, while highlighting the potential of structure-guided design to refine GlyT1-targeted therapies

An animal study showed that copper, via Atox1 as a transcription factor within the cell nucleus, binds to the Cyclin D1 promoter and promotes Cyclin D1 expression, thereby driving cell-cycle progression and cell proliferation (40)

Ryanodine receptor/calcium release channel PKA phosphorylation: a critical mediator of heart failure progression

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