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Results Oxidative stress in FH-deficient cells To elucidate the metabolic changes that occur on the loss of FH, we utilized the previously characterized mouse FH-deficient kidney cells ( Fh1 / ) and their wild-type (wt) isogenic control ( Fh1 fl/fl ) (ref

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Shuo Hu, [email protected] These authors have contributed equally to this work This article was submitted to Biomaterials, a section of the journal Frontiers in Bioengineering and Biotechnology Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

Instead, clinical success often depends on a balance between disease-relevant expression, pathway context, and the ability to achieve functional selectivity through dosing, delivery, or molecular mechanism

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