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This phenotype was manifested in the accumulation of lactic acid, pyruvate, and carnitine, increased alanine aminotransferase and aspartate aminotransferase levels in plasma [72], suppressed ETC activity, and reduced mitochondrial membrane potential ( m ) [79]

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To further characterise glucose homeostasis we subjected chow-fed lox/lox and HGKO mice to hyperinuslinaemiceuglycaemic clamps (Fig