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In chronic inflammatory diseases, the sustained activation of the lung-brain axis contributes to long-term symptoms, as exemplified in asthma, where airway inflammationwhich may be initiated or exacerbated by pollutant exposuredrives persistent vagal signaling through the NTS to functional regions such as the insula and prefrontal cortex, ultimately fostering neural phenotypes associated with dyspnea and mood disorders [331, 332]

Oxford: IRL Press

Half-Life is developed as part of the Moxie Incubator

CTR1 appears to play a primary role in this uptake, as indicated by research showing greatly reduced copper accumulation in the peripheral tissues of neonatal mice lacking CTR1 [17]