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ARID1A mutations, primarily nonsense or frameshift types, are more common in endometriotic epithelial cells and lead to reduced or absent ARID1A protein (108), These mutations are often linked to malignant transformation, as normal ARID1A expression is retained in non-cancerous endometriotic tissues (117)

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One consequence may be the existence of functional redundancy between isoforms making it difficult to study the biological functions of a particular gene using reverse-genetic approaches

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1 Introduction Multiple myeloma (MM), a hematologic malignancy characterized by clonal proliferation of bone marrow plasma cells, manifests clinically through CRAB criteria: hypercalcemia, renal dysfunction, anemia, and osteolytic lesions ( Ferroptosis, an iron-dependent non-apoptotic cell death driven by excessive lipid peroxidation, was first characterized by Dixon et al., in 2012 through studies on erastin and RAS-selective lethal compounds (Zhang et al., 2022