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The alignment between our in vitro results and the published observations in human post-mortem brains as well as vCSF of PD patients [46, 47], supports our findings and thus could be the basis for the development of TGFb-inhibiting drugs as therapeutic approach for PD and related synucleinopathies

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Patients with decreased saliva have trouble eating, speaking and are predisposed to oral infections and tooth decay

The redox homeostasis of cells is maintained by complex endogenous antioxidant defense systems, including endogenous antioxidant enzymes (catalase, superoxide dismutase, glutathione peroxidase, etc.) and nonenzymatic compounds (glutathione, metal-chelating proteins, etc.) [96]