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APAP overdose leads to excessive generation of the highly reactive metabolite n-acetyl-p-benzoquinone imine (NAPQI), which depletes glutathione (GSH), increases oxidative stress, and causes mitochondrial dysfunction, finally triggering immune response-mediated hepatotoxicity and massive hepatocytes necrosis [15, 16]

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Safety and efficacy of edaravone in well defined patients with amyotrophic lateral sclerosis: a randomized, double-blind, placebo-controlled trial

The UCP2/SIRT3/PGC-1 signaling axis, activated by matrine, plays an important role in limiting oxidative damage by decreasing mtROS production, inhibiting ferroptosis, and preserving mitochondrial function while also implicating peroxisomal metabolism in redox homeostasis (77)

Cochrane Database Syst Rev 2010(7) Sugiura H, Yoshida T, Tsuchiya K, Mitobe M, Nishimura S, Shirota S, Akiba T, Nihei H (2005) Klotho reduces apoptosis in experimental ischaemic acute renal failure