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Chronic valproic acid administration increases plasma, liver, and brain ammonia concentration and suppresses glutamine synthetase activity

Previous studies have found that BPC-157 did not exert a direct effect in terms of accelerating the cell proliferation of cultured tendon fibroblasts, 42 but our results suggested that BPC-157 modulates the cell viability and affects HUVEC cell cycle exit in G0/G1 phase

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Since IR is known to influence AD pathology, the ability of these hepatokines to impact both conditions suggests a multifaceted therapeutic potential for addressing metabolic and neurological dysfunctions