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10.1042/bsr20190681 11 HuangX.LiangR.LiuY.YuL.YangM.ShangB.et al (2024)

demonstrated that improving mitochondrial metabolism, reducing oxidative stress and mitochondrial fission via the deletion of DNA-dependent protein kinase catalytic subunit (DNA-PKcs), thus alleviating sepsis-mediated myocardial injury, liver injury, and kidney injury in a sepsis model

BCAT1E61A's higher enzymatic activity can promote the catabolism of BCAA and accelerate cell growth and metastasis, and BCAT1 can also activate the PI3K/AKT/mTOR pathway to contribute to the development of tumors [40]

Our GO term enrichment studies in the hepatocytes comparing BWS to nonBWS liver demonstrated an enrichment of fatty acid and lipid metabolic pathways in BWS hepatocytes, indicative of PPARA involvement in the dysregulation of these processes in BWS liver

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