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ImaiTInoueRKawadaYMoritaYInatomiONishidaAet alCharacterization of fungal dysbiosis in Japanese patients with inflammatory bowel disease

Assists in the absorption and utilization of glutathione and other nutrients 2

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While normal consumption of fructose feeds into glycogen biosynthesis (104), excessive fructose consumption suppresses fatty acid -oxidation (FAO) in the liver (105) and induces DNL by the induction of sterol regulatory element-binding protein-1 (SREBP1), acetyl-CoA carboxylase-1 (ACC1), and fatty acid synthase (FAS) (105, 106)

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