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Experimental models have been instrumental in elucidating how perturbations in circadian rhythms may foster a pro-tumorigenic environment, particularly through hormonal, immune, genetic, and epigenetic pathways 38

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D 1 (1 + 1 mixture of ergocalciferol and lumisterol), D 2 (ergocalciferol), D 3 (cholecalciferol), D 4 (22,23-dihydroergocalciferol) and D 5 (sitocalciferol) [134]