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Researchers reported the peptide was well-tolerated and observed changes in glycemic variability markers during the treatment period [3]

The disruption of copper homeostasis has increasingly been implicated in the pathogenesis of Alzheimer's disease (AD), a neurodegenerative disorder characterized by amyloid- (A) plaques, neurofibrillary tangles (NFTs) of hyperphosphorylated tau and neuronal loss (6,7)

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Treatment of high-altitude pulmonary edema by bed rest and supplemental oxygen

Mitochondrial oxidative DNA damage and exposure to particulate air pollution in mother-newborn pairs