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These results suggest that, following the activation of NF-B by docetaxel, apoptosis is elicited through a mitochondria-dependent pathway ( CTX is a taxane drug that can induce apoptosis or autophagy by inhibiting the phosphorylation of PI3K/Akt/mTOR and is effective in some drug-resistant tumors

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These systems improve pharmacological performance and align polyphenol-based strategies more closely with synthetic small-molecule and biologic therapies