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These data demonstrate that GSH synthesis in the liver is responsible for maintaining lipogenic gene expression and sustaining circulating triglyceride levels in vivo

The simulation applied the Ordinary Differential Equations (ODEs) to create an exploratory model by speculating the system progression and the experimental result [4] (Figure 5)

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Correction: Bone morphogenetic protein 4 alleviates nonalcoholic steatohepatitis by inhibiting hepatic ferroptosis

Bio-distribution Test In Vivo Fluorescent orid-liposome and TLR2 pep-orid-liposome were synthesized with rhodamine labeling for tracking purposes