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Mechanistically, MAGI3 enhances the interaction between GSK3 and -catenin by binding directly to the carboxyl terminus of -catenin, facilitating GSK3-mediated phosphorylation at the residues Ser33, Ser37, and Thr41 (Figs

Investigating the roles of oxidative stress, inflammation and genetic susceptibility contributes to a deeper understanding of NIHL pathophysiology, potentially leading to the development of pharmaceutical interventions, gene therapies, or innovative hearing protection devices

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In several of these models, BPC-157 reduced neurological deficits and sped up functional recovery

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