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However, many challenges and unknowns concerning how to modulate oxidative stress for further practical clinical applications exist, and more targeted research is needed

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Direct comparison of differentially expressed genes (DEGs) in our dataset to previously published datasets revealed that genes upregulated in amdh-1 mutants were also highly upregulated in skn-1(lax188) gain-of-function animals and downregulated in worms treated with skn-1 RNAi (fig 1f) (Nhan et al., 2019

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