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Their results for a wide range of solutesolvent size ratio are in good agreement with Ould-Kaddours work 36

Preclinical evidence suggests that partial transporter inhibition may be sufficient to increase extracellular glycine levels and enhance NMDA receptor activation, whereas excessive inhibition could disturb the finely regulated balance of co-agonist availability and synaptic signaling (Javitt 2012

[26], resulting in (1) underestimation of the impact of APAP on the incidence ofASD and (2) a lack of statistical power leading to confidence intervals that are too large to draw conclusions [2]

One explanation for this is attributed to the feedback regulation within the pathwaycontinuous NRF2 activation triggers the cellular program to express NRF2 transcriptional repressors (Figures 4A,B)