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Meanwhile, immunofluorescence (Figure 6B) results revealed that anwuligan treatment promoted the nuclear localization of Ctnnb1 in HFSCs, and this effect was inhibited by the Wnt pathway inhibitor XAV939

The above may account for some of the extracellular calreticulin, but does not fully explain why extracellular and cell membrane bound calreticulin are observed in other forms of cancers or in autoimmune patients (see Figure 3 and Table 1)

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