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Nevertheless, the vast majority of published studies rely on subcutaneous injection routes to ensure consistent systemic absorption and biological effect [11]

has received funding from the NIH (R01 AI-103173)

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because they were found to contain elevated levels of lead

This review integrates preclinical research related to the effects of both stress and antidepressant treatment on synaptic plasticity, and proposes normalization of synaptic function as a final common pathway for depression treatment