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Study Population This case-control study included patients aged 18 and over, hospitalized between January 2007 and December 2016 at Angers University Hospital, in the departments of Internal Medicine, Geriatrics or Neurology, and who had at least one plasma B12 testing

presented a heterozygous CHAC1 deletion mouse model that was protected from kidney disease progression across three different disease models, without any known phenotypic changes under control conditions ( Despite its injury-promoting effects in some organs, CHAC1 contributes to tissue repair in certain contexts

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Many people spend years addressing symptoms without recognizing that compromised gut integrity might be the underlying issue connecting their various concerns

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