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While TRT addresses low testosterone directly, peptides target complementary pathways growth hormone release, tissue repair, fat metabolism, and inflammation control creating a synergistic effect that neither therapy achieves as effectively on its own

Furthermore, HPV-infected cells adapt to OS situations by inhibiting OS-induced apoptosis and modulating antioxidant activity via its oncogenes, which bypasses DNA damage response pathways

These method-dependent outcomes may reflect mechanistic differences between mutational profiling methods (Supplementary Note)

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