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Hyperhomocyst (e) inemia as a risk factor for occlusive vascular disease

doi:10.1016/j.cell.2006.03.032

To evaluate the impact of ChP BMAL1 downregulation on astrocytic responses to amyloid plaques and on aquaporin 4 (AQP4) polarization, we performed GFAP staining to examine changes in astrocytes surrounding plaques (Fig

Protocol for the MPTP mouse model of Parkinsons disease

In parallel, excessive Cu + generated in GSH-rich reductive environments can bind lipoylated mitochondrial TCA-cycle proteins such as DLAT, inducing aberrant protein aggregation and triggering cuproptosis across multiple cancer types 187,189,192-194