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Rhus coriaria extract (RCE) exhibits notable effectiveness in fighting colorectal cancer cells by diminishing cell viability and colony formation, triggering Beclin-1-independent autophagy and caspase-7-dependent apoptosis, impacting the AKT/mTOR pathway, and influencing crucial proteins related to proteasome-mediated breakdown (87)

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Based on this mechanism of action alone, it is automatically assumed that these peptides contribute to tumor growth (or are at least correlated with this outcome)