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These findings suggest that TLR/PI3K/AKT-activated EPRS1 phosphorylation at Ser999 directs the first line of anti-inflammatory immune defense against inflammatory signals, including bacterial infection, while IFN-mediated EPRS1 phosphorylation at Ser999 shapes translational fine tuning to protect cells from inflammation and injury in the presence of persistent inflammatory stimuli

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Studies have shown that TSA downregulates extracellular matrix (ECM) gene transcription and collagen expression in dermal fibroblasts by inhibiting the TGF-/Smad and MAPK/ERK signaling pathways, contributing to its antifibrotic effects (60,61)

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