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Examining the results, IV-administered Co-60 was eliminated predominantly through the kidney, with a cumulative 5-day urinary excretion of 72.3% of the administered dose (FIG

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ACSL4 enhances the sensitivity of tumor cells to ferroptosis by modulating lipid composition [68], thereby inhibiting cell proliferation through ferroptosis [80]

This pathological process stems from excessive activation of glutamate receptors in neural cells and is considered a principal mechanism of neuronal death in various CNS disorders

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