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Another possible explanation is the hit-and-run hypothesis of viral oncogenesis, which posits that EBV may contribute to early oncogenic events but is not retained in the malignant clone [47, 48]

VV that have been bioengeneered to express IL-4

Conclusion: Our study provides a comprehensive metaproteomic analysis of bile samples related to gallstone disease, offering new insights into the microbiome-host interaction and gallstone formation mechanism

Steatosis may resolve with improved nutritional status and correction of trace mineral, vitamin, or fatty acid deficiencies

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