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Multiple human, animal, and cellular studies converge on the view that mitochondrial dysfunction, oxidative stress, and impaired mitophagy lead to leakage of mtDNA into the cytosol and extracellular space, where it activates innate immune sensors and sustains chronic inflammation

The bulk of BPC-157 research has focused on tissue repair (tendon, muscle, ligament, bone), gastrointestinal protection (ulcer models, IBD models, gut-brain axis), wound healing acceleration, and neuroprotection

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STAT5b is of most importance and directly regulates the expression of IGF-1 (43, 44), and was demonstrated to be a mediator of GH-induced IGF-1 production in rat granulosa cells (Figure 2) (33, 45, 46)