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the bacterial myrosinase in the intestine, rather than vegetable myrosinase, is responsible for the initial process of glucosinolate degradation

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T alone), but instead promoted a shift toward necrotic cell death (8.52 0.28%, p 0.05), despite a marginal trend toward increased B7-H3 transcripts in CSCs (Figure 9)

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SA also increased antioxidant enzyme expression via the Nrf2/keap1 pathway, enhanced autophagy through the AMPK-Akt/mTOR signaling pathway, and reduced intestinal fibrosis-associated proteins Collagen-I and -SMA