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In these patients, dysautonomia typically reflects a neurodegenerative disease

A role of reactive oxygen species in apoptotic activation of volume-sensitive Cl(-) channel

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Cell culture studies using adipocytes have shown that NNMT overexpression promotes lipid accumulation and insulin resistance, while NNMT knockdown or inhibition increases NAD+ levels, activates SIRT1, enhances mitochondrial respiration, promotes expression of thermogenic genes including UCP1, and drives white adipocyte browning toward a beige phenotype