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The following is a summary of documented tissue systems from peer-reviewed literature: This cross-tissue activity profile is directly attributable to the fact that its primary mechanisms VEGFR2-driven angiogenesis and NO system modulation are not tissue-specific

KD significantly reduced the release of pro-inflammatory cytokines TNF-, IL-1, and IL-6, while up-regulating the expression of anti-inflammatory cytokine IL-10 by inhibiting the over-activation of PI3K/Akt/NF-B signaling pathway

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Bellier J, Nokin MJ, Caprasse M, Tiamiou A, Blomme A, Scheijen JL, Koopmansch B, MacKay GM, Chiavarina B, Costanza B, et al

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