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WonSJYooBHBrennanAMShinBSKauppinenTMBermanAEet alEAAC1 gene deletion alters zinc homeostasis and exacerbates neuronal injury after transient cerebral ischemia

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Subsequently, the condensation recruits Z-RNA and induces ZBP1 recognition to Z-RNA, following active ZBP1 recruits RIPK3 within the SGs to form necrosomes, ultimately leading to the initiation of necroptosis [57]

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