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Therefore, patients that present with heightened inflammatory responses may have compromised cervicovaginal epithelial barriers that enable further spread of pathogens and antimicrobial resistance genes and increase their susceptibility towards resistant infections 93

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The gshR mutant can induce the formation of normal nodules, while the gpxA single and double mutants exhibited a nodulation phenotype coupled to more than 50% reduction in the nitrogen fixation capacity, these defects in nodulation were characterized by the formation of ineffective nodules

It also helps maintain the bodys natural antioxidant capacity by supporting glutathione synthesis and other redox pathways that protect cells during exercise and recovery