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Interaction between human-breast cancer metastasis and bone microenvir onment through activated hepatocyte growth factor/Met and beta-catenin /Wnt pathways

This approach significantly improved angiogenesis and accelerated wound repair in diabetic models, underscoring the role of mitochondrial dysfunction in NET-mediated pathology

This generally means that excess amounts are excreted in the urine, rather than being stored in the body, per NCI Dictionary of Cancer Terms

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