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M1, macrophage M1

One of the main ways that progressive tubular and interstitial fibrosis occurs is through apoptotic death and the buildup of ECM (37, 38)

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PMCID: PMC4733501

statins) which have demonstrated in vitro inhibition of neutrophil migration and epithelial cell production of chemoattractants and proteases and potentiation of macrophage efferocytosis.51,52 As efferocytosis appears to be involved in the pathogenesis of a variety of chronic lung and systemic inflammatory disease including BE, this would seem an attractive target for statins therapy.53 There are currently ongoing clinical trials to evaluate the effect of atorvastatin on patients with BE, with and without P