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Recent studies have shown IL-33 to be upregulated in epithelial cells and myofibroblasts in UC ( So far, most of these cytokines and chemokines have been studied more in mechanistic contexts than as clinical biomarkers

226 Cu deficiency can also alter the expression of myocardial gene profile, for example by upregulating the inflammatory cytokine TNF- and genes involved in regulating cardiac contractility, fibrosis, and calcium cycling, all of which may underlie the changes in cardiac function under Cu-deficient conditions

Since several ATGs and lysosomal cathepsin proteases depend on cysteines for their activities they can be the sensors or transducers of changes in the intracellular redox state through mechanisms involving redox signaling

Narita, Redistribution of the Lamin B1 genomic binding profile affects rearrangement of heterochromatic domains and SAHF formation during senescence

Based on these findings, Xinyu Zhou et al