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Although existing studies have tentatively confirmed the prevalence of dysregulated palmitoylation levels in inflammatory diseases, their dynamic regulatory mechanisms and pathological roles are limited, and the therapeutic agents for palmitoylation are still in their infancy, so more in-depth studies are needed to explore the clinical significance of palmitoylation modification in inflammatory diseases

The generated ROS, in turn, induces mitochondrial DNA damage, contributing to oxidative stress (42, 48)

GHK Peptide as a Natural Modulator of Multiple Cellular Pathways in Tissue Repair

Association between occupational exposure to pesticides and cardiovascular disease incidence: The Kuakini Honolulu Heart Program