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The red blood cell membrane cloaking served to extend nanoparticle circulation time and reduce immune recognition, whereas active propulsion enhanced dispersion and accumulation in the lungs

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Specifically, IL-5 priming led Siglec-8 cross-linking to cause a loss of mitochondrial membrane potential and production of ROS inside the eosinophil, which in turn drove the cell into apoptosis through a caspase-independent mechanism (183)