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Under pathological conditions, mitochondrial damage not only promotes translational activation of ATF4, but also induces transcriptional upregulation of the ROS-p38 MAPK-activating transcription factor 2 (ATF2) signaling axis driven by FGF-21 [102]

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In addition to MYC, mutations in the key gene KRAS, which are associated with c-MYC protein stability and activity, similarly result in dependence on exogenous glutamine for cell growth and proliferation

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