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This process is governed by BCL2 family proteins, including the pro-apoptotic BCL2 associated X, apoptosis regulator (BAX) or BCL2 antagonist/killer 1 (BAK1, also known as BAK) as well as anti-apoptotic members, such as BCL2 apoptosis regulator (BCL2) and BCL2 like 1 (BCL2L1, also known as BCL-XL)

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We also reported increased GSH levels during proliferation of rat hepatocytes such as plating primary cultures of rat hepatocytes under low density, which stimulates hepatocytes to shift from G 0 to G 1 phase of the cell cycle (Lu and Ge., 1992), and after 2/3 partial hepatectomy (PH) (Huang et al., 1998)

Paraesthesia, balance problems, or cognitive difficulties may persist despite normalising blood counts