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A precondition for inflammasome activation, the initial signal is produced by endogenous chemicals or microbes that cause NF-B to be activated, increasing the production of NLRP3 and proIL-1b (Zhang et al., 2014)

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Interestingly, relatively low concentrations of E2 are capable of robustly inhibiting germ cell apoptosis in cultured segments of human seminiferous tubules without survival factors (i.e., serum and hormones)